Search results (30)
« Back to PublicationsThe discovery of novel 8-azabicyclo[3.2.1]octan-3-yl)-3-(4-chlorophenyl) propanamides as vasopressin V1A receptor antagonists.
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Napier S. et al, (2011), Bioorg Med Chem Lett, 21, 3163 - 3167
Development of a novel high-throughput assay for the investigation of GlyT-1b neurotransmitter transporter function.
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Allan L. et al, (2006), Comb Chem High Throughput Screen, 9, 9 - 14
Investigating the potential role of oestrogen receptor beta (ER Beta) in affective disorders
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Irving NG. et al, (2006), JOURNAL OF AFFECTIVE DISORDERS, 91, S84 - S84
Fe65 and X11beta co-localize with and compete for binding to the amyloid precursor protein.
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Lau KF. et al, (2000), Neuroreport, 11, 3607 - 3610
Mint2/X11-like colocalizes with the Alzheimer's disease amyloid precursor protein and is associated with neuritic plaques in Alzheimer's disease.
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McLoughlin DM. et al, (1999), Eur J Neurosci, 11, 1988 - 1994
The Fe65 and X11 families of proteins: proteins that interact with the Alzheimer's disease amyloid precursor protein.
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McLoughlin DM. et al, (1998), Biochem Soc Trans, 26, 497 - 500
lzheimer's disease, amyotrophic lateral sclerosis, and transgenic mice.
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Bajaj NP. et al, (1998), J Neurol Neurosurg Psychiatry, 64, 711 - 715
Mapping of the human and murine X11-like genes (APBA2 and apba2), the murine Fe65 gene (Apbb1), and the human Fe65-like gene (APBB2): genes encoding phosphotyrosine-binding domain proteins that interact with the Alzheimer's disease amyloid precursor protein.
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Blanco G. et al, (1998), Mamm Genome, 9, 473 - 475
Cellular mechanisms regulating phosphorylation of the neuronal cytoskeleton in normal and disease states.
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Miller CCJ. et al, (1998), JOURNAL OF NEUROCHEMISTRY, 71, S55 - S55
Tau phosphorylation in transgenic mice expressing glycogen synthase kinase-3beta transgenes.
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Brownlees J. et al, (1997), Neuroreport, 8, 3251 - 3255
Overexpression of the mouse dishevelled-1 protein inhibits GSK-3beta-mediated phosphorylation of tau in transfected mammalian cells.
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Wagner U. et al, (1997), FEBS Lett, 411, 369 - 372
Tau phosphorylation in cells transfected with wild-type or an Alzheimer's disease mutant Presenilin 1.
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Irving NG. and Miller CC., (1997), Neurosci Lett, 222, 71 - 74
mouse model for Down syndrome exhibits learning and behaviour deficits.
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Reeves RH. et al, (1995), Nat Genet, 11, 177 - 184
BERRANT CYTOSKELETAL PHOSPHORYLATION AS A PATHOGENIC MECHANISM IN NEURODEGENERATIVE DISEASES - MECHANISMS OF CYTOSKELETAL PHOSPHORYLATION STUDIED IN CULTURED-CELLS AND TRANSGENIC ANIMALS
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MILLER CCJ. et al, (1995), JOURNAL OF CELLULAR BIOCHEMISTRY, 100 - 100
COMPARATIVE PHYSICAL, RECOMBINATIONAL AND BIOLOGICAL MAPPING OF MOUSE CHR16 AND HUMAN CHR21 (HSA21)
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REEVES RH. et al, (1995), CYTOGENETICS AND CELL GENETICS, 70, 180 - 181